Synvista Therapeutics Announces Published Data Demonstrating Elevation of Carboxymethyl-Lysine (CML) Is Associated with Decline
16 Dezembro 2008 - 11:30AM
PR Newswire (US)
Synvista Therapeutics Technology Provided CML Biomarker Data for
Studies MONTVALE, N.J., Dec. 16 /PRNewswire-FirstCall/ -- Synvista
Therapeutics, Inc. (NYSE Alternext US: SYI) today announced two
publications demonstrating findings from studies that indicated
that the presence of the advanced glycation end product (A.G.E.)
called carboxymethyl-lysine (CML) in older adults is associated
with a decline in renal function and arterial stiffness in older
adults. Kits developed through a license to Synvista's technology
were used in these studies. Synvista is validating this clinical
diagnostic assay as it prepares to submit a CML kit for 510(k)
clearance in the second half of 2009. "These studies represent an
important next step in our understanding of the role of CML in an
elderly population, and they provide physicians more information
that may be useful in the prevention of cardiovascular and renal
disease, particularly in older adults," said Richard D. Semba,
M.D., M.P.H, W. Richard Green Professor of Ophthalmology, The Johns
Hopkins University School of Medicine and lead author of both
studies. "As we have learned, the elevation of CML is preventable
in many cases, as it can be mitigated by changes in diet.
Therefore, to bring a clearer understanding of this A.G.E. to the
forefront may help doctors better explain to their patients how to
help prevent cardiovascular and renal complications." In one study,
published in a recent edition of American Journal of Hypertension,
investigators measured 493 adult subjects participating in the
Baltimore Longitudinal Study of Aging for their aortic pulse wave
velocity (an index of aortic stiffness) and CML (using Synvista's
proprietary technology). Results demonstrated that elevated levels
of CML in these subjects corresponded to increased arterial
stiffness, a known predictor of cardiovascular disease. The second
study, published in a recent edition of the European Journal of
Nutrition, demonstrated that the presence of CML in older adults is
independently associated with chronic kidney disease, and may be an
independent predictor of decline in renal function. This
population-based study of aging in a population from Tuscany,
Italy, followed 1,008 adults age 65 and over. At the conclusion of
the study after six months, there was a clear correlation between
those subjects with elevated CML levels and those that had renal
impairment, adjusting for those that already had chronic kidney
disease at the outset of the study. "These studies also clearly
demonstrate the utility of our technology to diagnose CML in older
adults. Like Dr. Semba, we firmly believe that an increased
understanding of CML in older adults will help doctors understand
the relationship between the presence of A.G.E.s and future
cardiovascular and renal disease, and help patients to understand
how to lower their CML levels to reduce cardiovascular and other
types of risk," said Noah Berkowitz, M.D., Ph.D., President and
Chief Executive Officer of Synvista Therapeutics. "We believe that
a clinical diagnostic test, upon clearance by the FDA, to measure
CML levels may one day provide these patients and their physicians
with a pathway for clinical intervention. We hope as a next step to
look at the relationship between cardiovascular outcome and
mortality." About CML The process of aging naturally increases the
presence of damaged proteins. Often this damage is caused by the
attachment of glucose to proteins in a process called glycation.
When the glucose-modified proteins go through further oxidation,
the resulting modified protein is described as an Advanced
Glycation End Product (A.G.E.). These damaged proteins are often
dysfunctional, inflammatory and damaging to organs such as the
heart, kidney and skin among others. Collagen (a protein normally
found in connective and structural tissue) is an example of a
protein that can be converted into an A.G.E. As a consequence of
advanced glycation and oxidation, collagen fibers can cross link,
which makes them less flexible. When formed in the heart and blood
vessels (the cardiovascular system), cross linked collagen induces
stiffness, a loss of adaptability and decreased reactivity which
makes the heart and blood vessels act less effectively and may
increase the risk of cardiovascular complications.
Carboxymethyl-lysine, or CML, is one highly common A.G.E. believed
to play a role in cardiovascular mortality, renal insufficiency,
increased aortic stiffness, and other clinical phenomena associated
with aging. Synvista Therapeutics is conducting clinical trials to
better understand the role of CML in age-related system decline.
Preliminary studies are revealing that CML is relevant to the cause
of loss in functionality of certain bodily systems (mainly the
cardiovascular and renal systems). It seems clear that the presence
and level of circulating CML may be considered a biomarker for the
likelihood of system decline. This is because CML in the blood is
similar to serum cholesterol biomarkers in that they can be lowered
by modifying diet or through drug intervention (e.g., use of
statins). Synvista's current research is designed to clarify the
relationship between A.G.E.s and chronic cardiovascular and renal
disease. About Synvista Therapeutics Synvista Therapeutics is a
biopharmaceutical company developing diagnostics and drugs to
diagnose, treat and prevent cardiovascular disease in people with
diabetes. The Company has developed a clinical diagnostic test for
Hp2-2 Diabetes. The genetic or protein form of this test can be
used to identify diabetic patients at high risk for cardiovascular
complications. These patients may benefit from a particular
formulation of vitamin E. The Company is also developing a kit to
measure CML (carboxy-methyllysine), another potential
cardiovascular risk marker. Synvista Therapeutics is developing
oral antioxidant drugs to treat the HDL dysfunction seen in Hp2-2
Diabetes, a disease affecting almost 7 million patients in the
United States. The Company is also developing alagebrium, a
proposed breaker of advanced glycation end products (A.G.E.s) for
the treatment of systolic and diastolic heart failure. Diastolic
heart failure represents a rapidly growing market of unmet medical
need, particularly common among diabetic patients. Alagebrium has
demonstrated relevant clinical activity in two Phase 2 clinical
trials in heart failure, as well as in animal models of heart
failure and nephropathy, among others. Alagebrium has been tested
in approximately 1,000 patients in multiple Phase 1 and Phase 2
clinical trials, allowing Synvista Therapeutics to assemble a
sizeable human safety database. For more information, please visit
the Company's Web site at http://www.synvista.com/. Any statements
contained in this press release that relate to future plans, events
or performance are forward-looking statements that involve risks
and uncertainties including, but not limited to, the risks
associated with the events described in this press release, the
Company's ability to obtain sufficient financial resources to
continue operations, future clinical development of Synvista
Therapeutics' diagnostic tests and product candidates, and other
risks identified in Synvista Therapeutics' filings with the
Securities and Exchange Commission. Further information on risks
faced by Synvista are detailed under the caption "Risk Factors" in
Synvista Therapeutics' Annual Report on Form 10-K for the year
ended December 31, 2007. These filings are available on a website
maintained by the Securities and Exchange Commission at
http://www.sec.gov/. The information contained in this press
release is accurate as of the date indicated. Actual results,
events or performance may differ materially. Synvista Therapeutics
undertakes no obligation to publicly release the result of any
revision to these forward- looking statements that may be made to
reflect events or circumstances after the date hereof or to reflect
the occurrence of unanticipated events. DATASOURCE: Synvista
Therapeutics, Inc. CONTACT: Synvista Therapeutics, Inc.,
+1-201-934-5000, or ; or Investor/Media Relations: Jules Abraham,
of Lippert/Heilshorn & Associates, +1-212-838-3777, or Web
Site: http://www.synvista.com/
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